文献类型：期刊文献

英文题名：Acetylsalicylic acid (ASA) induced fusarium rot resistance and suppressed neosolaniol production by elevation of ROS metabolism in muskmelon fruit

作者：Xue Huali[1,2];Sun Yaxuan[1];Li Lan[2];Bi Yang[3];Hussain Raza[4];Zhang Rui[3];Long Haitao[2];Nan Mina[2];Pu Lumei[2]

第一作者：Xue Huali

通讯作者：Xue, HL[1];Sun, YX[1];Xue, HL[2]

机构：[1]Beijing Union Univ, Beijing Key Lab Bioact Subst & Funct Foods, Beijing 100191, Peoples R China;[2]Gansu Agr Univ, Coll Sci, Lanzhou 730070, Peoples R China;[3]Gansu Agr Univ, Coll Food Sci & Engn, Lanzhou 730070, Peoples R China;[4]Univ Cent Punjab, Dept Food Sci & Technol, Fac Life Sci, 1 Khayaban E Jinnah Johar Town, Lahore, Pakistan

第一机构：北京联合大学应用文理学院|北京联合大学生物化学工程学院

通讯机构：[1]corresponding author), Beijing Union Univ, Beijing Key Lab Bioact Subst & Funct Foods, Beijing 100191, Peoples R China;[2]corresponding author), Gansu Agr Univ, Coll Sci, Lanzhou 730070, Peoples R China.|[1141726]北京联合大学生物化学工程学院;[11417]北京联合大学;[114172]北京联合大学应用文理学院;

年份：2020

卷号：265

外文期刊名：SCIENTIA HORTICULTURAE

收录：;Scopus(收录号：2-s2.0-85079170895);WOS:【SCI-EXPANDED(收录号:WOS:000522093500055)】；

基金：The work was financially supported by the Scientific Research Project from Facing Characteristic Discipline of Beijing Union University (Grant No. KYDE40201703) and Beijing Key Laboratory of Bioactive Substances and Functional Foods in Beijing Union University (Grant No. 12213991724010239).

语种：英文

外文关键词：Muskmelon; Acetylsalicylic acid; Trichothecenes; ROS metabolism; Fusarium rot

摘要：Major losses in the harvested muskmelon fruit can be attributed to decay fungi. Fusarium sulphureum is the major causal agent of fusarium rot in muskmelon, which in turn, not only causes quality deterioration but also leads to neosolaniol (NEO) contamination. Therefore, new strategies to manage muskmelon postharvest decay and decrease NEO production are of paramount importance. Acetylsalicylic acid (ASA), a derivative of salicylic acid (SA), is a putative endogenous signal molecular and can induce resistance against decay fungi in postharvest fruit and vegetable. To investigate the mechanism of induced resistance by ASA treatment against fusarium rot of muskmelon, the fruit was treated with ASA, followed by the inoculation with F. sulphureum. Results showed that the reduction of fusarium rot severity and the suppression of NEO accumulation were linked with the elevation of ROS metabolism. ASA treatment enhanced ROS accumulation and induced the enzymatic activities and upregulated gene expressions involved in ROS metabolism. Taken together, the present study provides substantial evidence which ASA induced resistance against F. sulphureum in muskmelon fruit to control the fusarium rot development.